RT Journal Article
T1 Pathology and diagnosis of brucellosis ( spp.) in veterinary species
A1 Rebollada Merino, Agustín Miguel
A1 Domínguez Rodríguez, Lucas José
A1 Rodríguez Bertos, Antonio Manuel
AB Brucellosis is an infectious disease that affects a wide range of animals, caused by bacteria belonging to 13 species and several biovars within the genus (, , , , , , , , , , , , , and unclassified strains). spp. infects small ruminants, cattle, swine, dogs, wild mammals, reptiles, amphibians, and fish, and most spp. have zoonotic potential. Characterization of lesions in animal brucellosis remains paramount in the diagnosis in endemic regions and even for countries free of brucellosis, where passive surveillance plays an indispensable role in upholding and sustaining their status. Pathology also contributes to understanding the pathogenesis and associating emerging pathogenic spp. with disease in animals. Here, we review the pathology of brucellosis in animals with an emphasis on cross-species transmission of "classical" spp. to nonpreferential hosts and "atypical" spp. We conclude that is an expansive genus identified in numerous animals, but there are still significant gaps in the knowledge of the pathology of brucellosis in unusual hosts and emerging spp. This is a great opportunity for veterinary pathologists to spearhead advancements in the knowledge of the pathogenesis and diagnosis of spp. infections. The isolation of a spp. and the correlation with lesions should follow molecular diagnostics to define the genetic signature of the isolate, providing a better understanding of epidemiology and contributing to the control of brucellosis across animal and human populations.
PB SAGE Publications
SN 0300-9858
YR 2026
FD 2026
LK https://hdl.handle.net/20.500.14352/135010
UL https://hdl.handle.net/20.500.14352/135010
LA eng
NO 1. Rebollada-Merino A, Domínguez L, Rodríguez-Bertos A. Pathology and diagnosis of brucellosis (Brucella spp.) in veterinary species. Veterinary Pathology. 2026;0(0). doi:10.1177/03009858261435106
NO Justificación de autores:The manuscript was written by ARM with contributions from the other authors.
DS Docta Complutense
RD 27 abr 2026