TY  - JOUR
AU  - Guo, Feifei
AU  - Estevez Vázquez, Olga
AU  - Benedé Ubieto, Raquel
AU  - Maya Mile, Douglas
AU  - Zheng, Kang
AU  - Gallego Durán, Rocío
AU  - Rojas Ávalos, Ángela
AU  - Ampuero, Javier
AU  - Romero Gómez, Manuel
AU  - Philip, Kaye
AU  - Egbuniwe, Isioma U.
AU  - Chen, Chaobo
AU  - Simon, Jorge
AU  - Delgado, Teresa C.
AU  - Martínez Chantar, Maria L.
AU  - Sun, Jie
AU  - Reissing, Johanna
AU  - Bruns, Tony
AU  - Lamas Paz, Arantza
AU  - Woitok, Marius Maximilian
AU  - Regueiro, José R.
AU  - Liedtke, Christian
AU  - Trautwein, Christian
AU  - Bañares Cañizares, Rafael
AU  - Cubero Palero, Francisco Javier
AU  - Benede Ubieto, Raquel
AU  - Gómez Del Moral Martín-Consuegra, Manuel María
AU  - Vaquero Martín, Francisco Javier
AU  - Nevzorova, Yulia
PY  - 2021
DO  - 10.3390/cancers14010192
SN  - 2072-6694
UR  - https://hdl.handle.net/20.500.14352/71932
T2  - Cancers
AB  - Background: Metabolic-associated fatty liver disease (MAFLD) has risen as one of the leading etiologies for hepatocellular carcinoma (HCC). Oncogenes have been suggested to be responsible for the high risk of MAFLD-related HCC. We analyzed the impact...
LA  - eng
M2  - 1
PB  - MDPI
KW  - metabolic-associated fatty liver disease (MAFLD)
KW  - c-myc
KW  - oncogene
KW  - tumorigenesis
KW  - metformin
TI  - A Shortcut from Metabolic-Associated Fatty Liver Disease (MAFLD) to Hepatocellular Carcinoma (HCC): c-MYC a Promising Target for Preventative Strategies and Individualized Therapy
TY  - journal article
VL  - 14
ER  -